Study on the antidepressant mechanism of ginseng-fragrant solomonseal rhizome couplet medicines on inhibiting the activation of inflammasomes NLRP1, NLRC4, and AIM2, and regulating the expression of inflammatory cytokines
Experimental Studies|更新时间:2024-08-02
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Study on the antidepressant mechanism of ginseng-fragrant solomonseal rhizome couplet medicines on inhibiting the activation of inflammasomes NLRP1, NLRC4, and AIM2, and regulating the expression of inflammatory cytokines
“The latest research has found that ginseng jade bamboo medicine can effectively improve depressive like behavior in chronic stress rats, and its antidepressant effect may be related to the inhibition of cortical inflammasome activation and its mediated inflammatory response.”
Journal of Beijing University of Traditional Chinese MedicineVol. 47, Issue 7, Pages: 939-947(2024)
作者机构:
辽宁中医药大学 沈阳 110032
作者简介:
QIAO Tie, Ph.D., Researcher, Master′s Supervisor. Liaoning University of Traditional Chinese Medicine, No. 79, Chongshan Donglu Road, Huanggu District, Shenyang 110032. E-mail: qiao_0702@163.com
基金信息:
Postdoctoral Science Foundation of China(2021MD703843)
ZHANG Huayu, CAO Jialu, ZHENG Bingyuan, et al. Study on the antidepressant mechanism of ginseng-fragrant solomonseal rhizome couplet medicines on inhibiting the activation of inflammasomes NLRP1, NLRC4, and AIM2, and regulating the expression of inflammatory cytokines[J]. Journal of beijing university of traditional chinese medicine, 2024, 47(7): 939-947.
DOI:
ZHANG Huayu, CAO Jialu, ZHENG Bingyuan, et al. Study on the antidepressant mechanism of ginseng-fragrant solomonseal rhizome couplet medicines on inhibiting the activation of inflammasomes NLRP1, NLRC4, and AIM2, and regulating the expression of inflammatory cytokines[J]. Journal of beijing university of traditional chinese medicine, 2024, 47(7): 939-947. DOI: 10.3969/j.issn.1006-2157.2024.07.009.
Study on the antidepressant mechanism of ginseng-fragrant solomonseal rhizome couplet medicines on inhibiting the activation of inflammasomes NLRP1, NLRC4, and AIM2, and regulating the expression of inflammatory cytokines
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